A new fungus identified in Germany in 2019 is causing a concerning number of sexually transmitted cases of ringworm across the US and Europe.1 Trichophyton mentagrophytes genotype VII, known as TMVII (and called TM 7), is a dermatophyte that was first noted when it infected a group of people returning from sex tourism trips in Southeast Asia. It has been spreading among men who have sex with men (MSM) in France since 2021. The first case in the US was reported in June 2024.2 If prevention isn't prioritized and the knowledge gap about the fungus isn’t narrowed, there is a risk of an epidemic. A small survey conducted by the CDC found that many infectious disease specialists were unaware of TMVII, and few knew how to treat it.3
Dermatophytes are the most common cause of superficial fungal infections worldwide.4 TMVII is a dermatophyte, and like all dermatophytes, it infects keratinized tissues such as skin, hair, and nails. TMVII is of the strain T mentagrophytes. Its internal transcribed spacer (ITS) genotype distinguishes it from other T mentagrophytes strains and can only be detected by genomic sequencing.5
While classic dermatophytoses are typically transmitted via fomites, shared surfaces, or animal contact, TMVII is transmitted primarily through sexual contact.1
In 2019, TMVII became considered a sexually transmitted infection (STI) pathogen in Germany, following a number of tinea genitalis cases associated with sexual contact.2 Some other cases were confirmed in Italy, Spain, and the United Kingdom. Cases in China linked to rabbit farming raise concern over zoonotic transmission potential.3
The first US case involved a patient who had a history of sexual contact with multiple partners during travel to Europe and California.6 Many other cases were recorded, and by early 2026, Minnesota alone reported more than 30 cases.7 A recently published systematic review of published cases found that most of the patients were male, from the men having sex with men (MSM) key population.8
TMVII is highly infectious, and it is transmitted via direct skin-to-skin contact during sexual activity. Most patients with it have a history of sexual exposure, with lesions consistently found around the genital and perianal region.4Symptoms can show up anywhere from a few days to weeks after exposure. Individuals without any visible lesions may still shed enough fungus to transmit it but the exact risk from assymptomatic spread is unclear.9 Immune suppression may increase the likelihood that an individual will come down with it upon exposure. Infected individuals are advised to keep away from sexual contact until lesions are fully healed.
Top 3 Takeaways
- Recognition can be difficult. TMVII may resemble eczema, psoriasis, or other infections, making persistent genital, perianal, or facial lesions important diagnostic clues.
- Confirming the genotype requires specialized testing. KOH microscopy can support a fungal diagnosis but cannot identify TMVII specifically.
- Follow-up and counseling matter. Treatment may be prolonged, and patients need clear guidance on medication adherence, avoiding contact with affected skin, and notifying partners.
Even though TMVII causes fungal infections that resemble infections caused by other dermatophytes, TMVII lesions are more inflammatory, extensive, and harder to treat. The most infected parts of the body include the genitals (tinea pubogenitalis), buttocks (tinea glutealis), face (tinea faciei), and trunk or limbs (tinea corporis).6 The most common kind is tinea pubogenitalis.
TMVII lesions have well-defined borders. They are also red, scaly, itchy, and sometimes painful. They can be mistaken for eczema or psoriasis, making diagnosis confusing.10
In Berlin, a cohort study of 51 confirmed TMVII patients found that the median time of diagnosis was long, at 42.5 days, with misdiagnosis and concomitant improper treatment occurring commonly.10
“The emergence and spread of sexually transmitted T mentagrophytes genotype VII (TMVII) may be driven in part by delayed or incorrect diagnosis and inappropriate corticosteroid use. These factors can suppress inflammation without treating the underlying fungal infection, allowing it to persist, worsen, and spread to additional sites in the body or to other individuals. Greater awareness among clinicians is important to facilitate early recognition and appropriate treatment,” says Rosa M. Andrade, MD, a board-certified physician specializing in infectious diseases at UCI Health.