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When disease-causing invaders like bacteria infect a human host, cells of various types swing into action, coordinating their activities to address the threat. In new research appearing in this months issue of the journal Nature Immunology, Roy Curtiss, director of the Center for Infectious Diseases and Vaccinology at the Biodesign Institute at Arizona State University, along with international collaborators, investigates the coordination of a particular type of immune response, involving the release of of IFN- a cell-signaling protein molecule known as a cytokine.


















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